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For HPV, t would be appropriate to consider supporting the parts of the immune response that are particularly importantly involved in anti-viral surveillance, which would be support for the Th1 response (typically done with berberine, baicalin, ginger, and sulforaphane), and support for NK cell response (typically done with astrgalus, andrographis, and reishi aka ganoderma). Also supporting methylation is essential !
Aside from systemic treatment, Ive heard of people using interferon that is given vaginally. it’s interferon alpha-2. And often people use Epigen spray 0,1%.
With any cancer risk, You want to support for Th1 response and NK cell response. Direct antiviral measures would also be very suitable for HPV. In that regard, licorice (which is what the Epigen MS refers to is based on, for example) can be of substantial utility, as would other natural substances like vitamin A. It’s also noteworthy that viral replication includes an NFkB dependent step, so some amount of downregulation of NFkB would be suitable to consider. The tricky part there is that too much of this would engender a tolerogenic shift, so you don’t want to flood the person with curcumin. You could consider 500mg tid, but watch the TGFb level to see if you’re pushing TGFb up.
See my other comments about TGFb in one of the other threads to get a sense of the balance there and of GSH related inhibition of TGFb. I would strongly consider giving GSH glutathione and not giving NAC, to avoid NAC fueling an increase in tolerogenic TGFb levels. You don’t want a lot of tolerogenic signaling in a case like this. If you are taking NAC, watch the lab TGFb levels because if they go up, you can tip towards Th2 dominance.
Another risk factor for persistent high-risk HPV infections and dysplasia/cervical cancer is dysbiosis in the vaginal microbiome. This actually is a risk factor for other STIs as well. vaginal probiotic suppositories can be helpful.
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